It’s a widespread chronic pain for both men and women, the increasing rate of this depression in people is creating tension as the time passes.
Some people think that this condition is not reversible.
Recent researches have proved this theory wrong and have brought the real truth in front of us.
So, from the beginning of any pain, the victim must beware of this problem and see a doctor as soon as possible.
Gout is one of the most painful forms of arthritis. It occurs when too much uric acid accumulates in the body.
This accumulation can lead to:
- Deposits of crystals of uric acid in the joints or joints, and often accumulate in the big toe.
- Deposits of uric acid (called gouty tophi) that look like lumps under the skin.
- Kidney stones (stones) due to the crystals of uric acid in the kidneys.
Introduction of Gout
This condition is a complication of arthritis. The unbearable pain in the toe base is called “Gout.” A conventional is not a new generation disease.
The inside parts of the bones cause an obstacle in blood circulation. We all know that blood circulation is a continuous process. So, any problem in this process will result in gout.
Now the question is, why gout causes the most pain in the toe base?
Well, the answer is straightforward. It is not necessary to be an expert on the human body to find the reason.
A human passes most of his time walking and running. The toes have a significant role in this procedure. That is why any interruption will cause problems in the toes.
Causes
The cause of the Gout is an excessive accumulation of uric acid in the body. Uric acid comes from the decomposition of substances called purines.
Medical experts can find purines in all tissues of the body. They can also see them in many foods such as liver, dried beans, and peas and anchovies.
Usually, uric acid dissolves in the blood, passes through the kidneys and leaves the body in the urine.
However, uric acid can build up in the blood when:
- The body increases the amount of uric acid it produces.
- The kidneys do not remove enough uric acid.
- Many foods rich in purines get consumed.
When the uric acid concentration in the blood is high, it is known as hyperuricemia.
Most people with hyperuricemia do not get the Gout. However, it can occur if crystals of uric acid get formed in the body in excessive amounts.
A person is more likely to have gout if:
- He has relatives with this disease.
- He’s a male.
- Have overweight.
- Drink too much alcohol.
- Eat too many foods rich in purines.
- It has a defective enzyme that makes it difficult for the body to decompose the purines.
- Exposed to lead in the environment.
- Have had an organ transplant.
- Take medications such as diuretics, aspirin, cyclosporine or levodopa.
- Take the vitamin niacin.
The First Step
After finding this disease in bones, we must see a doctor as soon as possible. There are treatments efficient enough to take this pain away.
The curing process will start from physiotherapy. This therapy involves massages and medication. The low powered steroid is a conventional medicine for gout.
If the pain is too severe, then the patient should be diagnosed in a hospital and show the results to a specialist.
How is gout diagnosed?
The doctor will ask about the symptoms, personal and family history of gout. Signs and symptoms of gout include:
- Hyperuricemia (high concentrations of uric acid in the blood).
- Crystals of uric acid in joint fluid.
- More than one attack of acute arthritis.
- Arthritis that occurs in one day and causes swelling, redness, and heat in the joint.
- Attack of arthritis in a single joint, usually in the big toe, ankle or knee.
The doctor can take a sample of fluid from an inflamed joint to see if any crystals are associated with gout to confirm this diagnosis.
Cure for Gout
Some specialized therapy centers give proper medicine to patients.
Sauna is becoming popular as a beautification process. But the fact is that is helpful in gout. The Low powered steroid will provide for temporary relief.
Doctors use different types of medications to treat an acute gout attack that includes:
- Non-steroidal anti-inflammatory drugs (NSAIDs).
- Corticosteroids, such as prednisone.
- Colchicine, which works best when taken within the first 12 hours of an acute attack.
- Sometimes, doctors prescribe NSAIDs or colchicine in small amounts daily to prevent future attacks. There are also medicines that reduce the concentration of uric acid in the blood.
Prevention of Gout
Well, gout can be controlled in a very processed way. First of all, walking with a conventional technique will decrease the upside pressure on the body.
Running on the grass or treadmill is beneficial techniques. Every person should tell their doctor about the pain on a regular basis.
While working or walking, a short pause can gradually banish the pain. Again, the prescription should get authenticated by a certified doctor.
The physician should check the conditions of gout and adequately medicate the patient.
The upside discussion can contribute to finding out and prevent the symptoms of gout.
What can people with gout do to stay healthy?
Some things people can do to stay healthy are:
- Take the medicines as ordered by the doctor.
- Tell the doctor all the medicines and vitamins you are taking.
- Schedule follow-up visits with your doctor.
- Maintain a healthy and balanced diet. Avoid foods that are rich in purines and drink plenty of water.
- Exercise regularly and maintain a healthy weight. Ask your doctor how you can lose weight safely. Rapid and extreme weight loss can increase the concentration of uric acid in the blood.
What research is being done on gout?
Researchers are studying:
- The NSAIDs effectiveness for the treatment of gout.
- The optimal dose of gout medications.
- New harmless drugs that decrease the concentration of uric acid in the blood and reduce symptoms.
- New treatments to block a chemical called tumor necrosis factor.
- Enzymes that break down purines in the body.
- The function of foods and certain vitamins.
- The impact of genetic factors and the environment.
- The interaction of the cells involved in acute attacks of gout.